Theobromine – is the primary methylxanthine found in products of the cocoa tree, Theobroma cocao. As a member of the methylxanthine family, it is thought to elevate levels of serotonin, the same action as the popular anti-depressants. Theobromine has a lot of research that shows its extraordinary effects on fat loss, appetite suppression, and mobilization of fatty deposits. Theobromine acts as a mild diuretic and stimulant, which creates a synergistic effect with caffeine.

Phenylethylamine HCL – this amazing compound is probably the cleanest stimulant ever researched, and it is naturally present in human fluids and tissues. Although categorized as a stimulant, it has the remarkable ability to simulate the central nervous system without causing nervousness or the jittery feeling. Phenylethylamine is found in chocolate and is responsible for its effects on mood, appetite, and sense of well-being. Until recent discoveries, PEA, or phenylethylamine, was rapidly destroyed within the body. If included with novel delivery systems, phenylethylamine HCL is provided “safe transport” and this metabolic fate is avoided and pharmacological activity becomes extremely apparent. This catecholamine precursor is responsible for elevating the metabolic rate and promoting a sense of satiety. Phenylethylamine acts on alpha-receptors in the brain, as do norepinephrine and certain prescription anti-obesity drugs. It is also believed to cause the release of dopamine in the pleasure sensing areas of the brain. Phenylethylamine HCL has a close chemical relationship to pharmaceutical stimulants, because it is the “backbone” of many pharmaceutical compounds.

Yohimbine HCL – has been shown in many clinical trials to effectively block alpha 2 adrenoreceptors. These studies have found that yohimbine increases the amount of non-esterfied fatty acids (NEFA’s) a product of lipolysis (the breakdown of fat), in the blood-stream for both lean and overweight subjects. There are a number of feedback mechanisms that prevent the release of norepinephrine (NE), one of the body’s primary lipolytic hormones. When NE is released, such as when taking methylsynephrine and acacia rigidula, it stimulates both the alpha and beta adrenoreceptors. Stimulation of the beta adrenoreceptors has the opposite effect, preventing the release of NE and lipolysis. Yohimbine prevents this negative feedback mechanism, and works in a synergistic fashion with the other components to increase NE and lipolysis. There are a number of reasons why alpha-2 inhibition is specifically useful. First, while the beta-adrenergic system primarily controls lipolysis during periods of intense activity, during rest, which makes up most of our day, the alpha-adrenergic system is in control. Also, “stubborn fat” areas – usually the abdominal area in men and the glutofemoral area in women – contain a higher ratio of alpha-2 receptors, making yohimbine particularly effective in these areas (whereas other drugs that increase NE may be somewhat counterproductive). Finally, alpha-2 blockade increases blood flow in adipose tissue, which prevents fat from being retained in the area.

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